DAAs work by inhibiting specific proteins or enzymes critical for the viral life cycle. For example, in the context of Hepatitis C, DAAs can target proteins such as NS3/4A protease, NS5A, and NS5B RNA-dependent RNA polymerase. By binding to these viral proteins, DAAs prevent the virus from completing its replication cycle, leading to a significant reduction in viral load and, in many cases, complete eradication of the virus from the host.
