Uday Bandyopadhyay - Infectious Diseases

Author Information

Uday Bandyopadhyay is a distinguished researcher affiliated with the Division of Infectious Diseases and Immunology at the CSIR-Indian Institute of Chemical Biology, located in West Bengal, India. His research focuses on various aspects of infectious diseases, pharmacology, and cellular mechanisms, contributing significantly to our understanding of these fields.

Research Contributions

Uday Bandyopadhyay has made substantial contributions to the study of infectious diseases and related fields. His research encompasses a wide range of topics including the impact of non-steroidal anti-inflammatory drugs (NSAIDs) on organ damage, the role of Rab7 in the assembly of retromer complexes in Plasmodium falciparum, and the regulation of lung fibrosis by sirtuins. Additionally, his work has explored mitochondrial quality control mechanisms and their implications in various pathophysiological conditions. His recent publications indicate a strong interest in malaria research and the development of chemotypes targeting drug-resistant strains.

Aliases

Uday Bandyopadhyay is also known by the aliases U. Bandyopadhyay and U Bandyopadhyay in academic publications.

Publication and Citation Metrics

Metric	Value
Total Papers	94
Total Citations	8471
H-index	41

Publications:

Non-steroidal anti-inflammatory drugs (NSAIDs) and organ damage: A current perspective

DOI: 10.1016/j.bcp.2020.114147

Year: 2020

Rab7 of Plasmodium falciparum is involved in its retromer complex assembly near the digestive vacuole.

DOI: 10.1016/j.bbagen.2020.129656

Year: 2020

Sirtuins as endogenous regulators of lung fibrosis: A current perspective.

DOI: 10.1016/j.lfs.2020.118201

Year: 2020

Mediators of mitophagy that regulate mitochondrial quality control play crucial role in diverse pathophysiology.

DOI: 10.1007/s10565-020-09561-1

Year: 2020

NOBEL PRIZE IN PHYSIOLOGY OR MEDICINE

DOI: 10.36094/sc.v86.nobel_prize_physiology_or_medicine.bandyopadhyay.390

Year: 2020

Hydrazonophenol, a Food Vacuole-Targeted and Ferriprotoporphyrin IX-Interacting Chemotype Prevents Drug-Resistant Malaria.

DOI: 10.1021/acsinfecdis.8b00178

Year: 2019

Correction: Impact of intravascular hemolysis in malaria on liver dysfunction: Involvement of hepatic free heme overload, NF-κB activation, and neutrophil infiltration.

DOI: 10.1074/jbc.AAC119.012033

Year: 2019

Withdrawal: Tryptamine-gallic acid hybrid prevents non-steroidal anti-inflammatory drug-induced gastropathy: Correction of mitochondrial dysfunction and inhibition of apoptosis in gastric mucosal cells.

DOI: 10.1074/jbc.W119.012035

Year: 2019

Indomethacin impairs mitochondrial dynamics by activating the PKCζ–p38–DRP1 pathway and inducing apoptosis in gastric cancer and normal mucosal cells

DOI: 10.1074/jbc.RA118.004415

Year: 2019

Withdrawal: Lansoprazole protects and heals gastric mucosa from non-steroidal anti-inflammatory drug (NSAID)-induced gastropathy by inhibiting mitochondrial as well as Fas-mediated death pathways with concurrent induction of mucosal cell renewal.

DOI: 10.1074/jbc.W119.012034

Year: 2019

How Can Adverse Effects be Minimized?

Are There Concerns About Antiviral Resistance?

What Role Do Vaccines Play?

How Does Inherited Immunity Work?

How Can Hepatitis B be Prevented?

How Are These Diseases Diagnosed?

Why Are Infectious Diseases a Global Health Concern?

How Can Genomics Improve Diagnostics?

What is the Emergency Use Authorization (EUA)?

What are Common Methods for Symptomatic Relief?

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