michinaga ogawa - Infectious Diseases

Author Information

Michinaga Ogawa is a researcher affiliated with the Department of Bacteriology I at the National Institute of Infectious Diseases, located in Shinjuku-ku, Tokyo, Japan. His work primarily focuses on infectious diseases, with a special interest in the mechanisms of autophagy in response to bacterial infections.

Research Contributions

Michinaga Ogawa has made significant contributions to the understanding of autophagy in the context of infectious diseases. His research has explored the interactions between the immune system and pathogens like Hepatitis B virus and Streptococcus pneumoniae. Some of his notable works include studies on how these pathogens manipulate host autophagy processes to enhance their own survival and replication. His research on selective autophagy and the molecular mechanisms involving bacterial and viral infections has provided valuable insights into potential therapeutic targets.

Aliases

Michinaga Ogawa is known by the same name in his academic and research publications.

Publication and Citation Metrics

Metric	Value
Total Papers	77
Total Citations	15,171
h-index	38

Publications:

Crosstalk between the innate immune system and selective autophagy in hepatitis B virus infection.

DOI: 10.1080/15548627.2022.2059747

Year: 2022

Galectin-9 restricts hepatitis B virus replication via p62/SQSTM1-mediated selective autophagy of viral core proteins

DOI: 10.1038/s41467-022-28171-5

Year: 2022

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

DOI: 10.1080/15548627.2020.1797280

Year: 2021

The multi-step mechanism and biological role of noncanonical autophagy targeting Streptococcus pneumoniae during the early stages of infection

DOI: 10.1080/15548627.2020.1743937

Year: 2020

Streptococcus pneumoniae triggers hierarchical autophagy through reprogramming of LAPosome-like vesicles via NDP52-delocalization

DOI: 10.1038/s42003-020-0753-3

Year: 2020

Streptococcus pneumoniae hijacks host autophagy by deploying CbpC as a decoy for Atg14 depletion

DOI: 10.15252/embr.201949232

Year: 2020

Streptococcus pneumoniae promotes its own survival via choline-binding protein CbpC-mediated degradation of ATG14.

DOI: 10.1080/15548627.2020.1776475

Year: 2020

Molecular mechanisms of Streptococcus pneumoniae‐targeted autophagy via pneumolysin, Golgi‐resident Rab41, and Nedd4‐1‐mediated K63‐linked ubiquitination

DOI: 10.1111/cmi.12846

Year: 2018

Bacterial secretion system skews the fate of Legionella-containing vacuoles towards LC3-associated phagocytosis

DOI: 10.1038/srep44795

Year: 2017

Intracellular fate of Ureaplasma parvum entrapped by host cellular autophagy

DOI: 10.1002/mbo3.441

Year: 2017

What is Pink Eye?

What Preventive Measures Can Be Taken?

How Important is International Collaboration?

What Are Some Examples of Inherited Immunity?

How is HSV Transmitted?

How Can Couples Stay Safe in a Monogamous Relationship?

Why Is Global Cooperation Important?

What are the Risk Factors for Developing Dengue Hemorrhagic Fever?

What are the Challenges?

What Are Blood Tests?

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