Jérémie Rosain - Infectious Diseases

Author Information

Jérémie Rosain is a researcher affiliated with the Laboratory of Human Genetics of Infectious Diseases at the Necker Branch of Inserm U1163, located at Necker Hospital for Sick Children in Paris, France. His work primarily focuses on the genetic, immunological, and clinical aspects of infectious diseases, with particular attention to conditions involving immune deficiencies and autoimmunity.

Research Contributions

Jérémie Rosain has made significant contributions to understanding the role of autoantibodies and genetic deficiencies in infectious diseases. His research has explored the impact of autoantibodies neutralizing Type I Interferons, particularly in the context of COVID-19, where such antibodies have been linked to increased mortality. Additionally, his work covers various genetic deficiencies, such as IRF8 deficiency and STAT1 deficiency, which contribute to complex immunological conditions. His studies have advanced the understanding of rare immunodeficiencies and their implications for infectious disease susceptibility and outcomes.

Aliases

Jérémie Rosain is also known by several aliases, including J Rosain, J. Rosain, and Jeremie Rosain. These variations are commonly used in academic publications and databases.

Publication and Citation Metrics

Metric	Value
Total Papers	86
Total Citations	6499
h-index	28

Publications:

Autoantibodies Neutralizing Type I Interferons in 20% of COVID-19 Deaths in a French Hospital

DOI: 10.1007/s10875-021-01203-3

Year: 2022

Pulmonary Alveolar Proteinosis and Multiple Infectious Diseases in a Child with Autosomal Recessive Complete IRF8 Deficiency

DOI: 10.1007/s10875-022-01250-4

Year: 2022

A partial form of inherited human USP18 deficiency underlies infection and inflammation.

DOI: 10.1084/jem.20211273

Year: 2022

The risk of COVID-19 death is much greater and age-dependent with type I IFN autoantibodies

DOI: 10.21203/rs.3.rs-1225906/v1

Year: 2022

Autoantibodies against Type I IFNs in Patients with Ph-negative Myeloproliferative Neoplasms.

DOI: 10.1182/blood.2021014890

Year: 2022

Genetic, Immunological, and Clinical Features of 32 Patients with Autosomal Recessive STAT1 Deficiency

DOI: 10.4049/jimmunol.2001451

Year: 2021

Biochemically deleterious human NFKB1 variants underlie an autosomal dominant form of common variable immunodeficiency.

DOI: 10.1084/jem.20210566

Year: 2021

Inherited GATA2 Deficiency Is Dominant by Haploinsufficiency and Displays Incomplete Clinical Penetrance

DOI: 10.1007/s10875-020-00930-3

Year: 2021

High Th2 cytokine levels and upper airway inflammation in human inherited T-bet deficiency

DOI: 10.1084/jem.20202726

Year: 2021

Autoantibodies neutralizing type I IFNs are present in ~ 4% of uninfected individuals over 70 years and account for ~ 20% of COVID-19 deaths

DOI: 10.1126/sciimmunol.abl4340

Year: 2021

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