Egon Demetz - Infectious Diseases

Author Information

Egon Demetz is affiliated with the Department of Internal Medicine II, specializing in Infectious Diseases, Pneumology, and Rheumatology at the Medical University of Innsbruck, located in Innsbruck, Austria. His research primarily focuses on understanding the complex interactions between metabolic processes, immune responses, and infectious diseases.

Research Contributions

Egon Demetz has made significant contributions to the field of infectious diseases, with an emphasis on the interplay between iron metabolism and immune function. His research has explored various aspects of how iron deficiency and overload affect cellular processes and immune responses. Notably, his work includes studies on mitochondrial function in the context of iron deficiency anemia, the role of Toll-like receptors in osteoblastic phenotype switching, and the impact of lipid homeostasis on bacterial infections. He has also investigated the mechanisms of hyperinflammation in Salmonella infections and the regulation of immune responses in viral infections such as COVID-19 and influenza.

Aliases

Throughout his academic career, Egon Demetz has been recognized under the aliases "E. Demetz" and "Egon Demetz". These variations are used interchangeably in scientific publications and databases.

Publication and Citation Metrics

Metric	Count
Total Citation Count	1693
h-index	23
Total Paper Count	63

Publications:

Mitochondrial Respiration in Response to Iron Deficiency Anemia: Comparison of Peripheral Blood Mononuclear Cells and Liver

DOI: 10.3390/metabo12030270

Year: 2022

O7 Toll-like receptor 3 mediates osteoblastic phenotype switch in calcific aortic valve disease

DOI: 10.1093/bjs/znab282.012

Year: 2021

Lipid dyshomeostasis and the control of bacterial infection by macrophages

DOI: 10.1016/j.atherosclerosis.2021.06.222

Year: 2021

The Role of Innate Immunity and Bioactive Lipid Mediators in COVID-19 and Influenza

DOI: 10.3389/fphys.2021.688946

Year: 2021

Ferritin H deficiency deteriorates cellular iron handling and worsens Salmonella typhimurium infection by triggering hyperinflammation

DOI: 10.1172/jci.insight.141760

Year: 2021

Lysis reagents, cell numbers, and calculation method influence high-throughput measurement of HDL-mediated cholesterol efflux capacity

DOI: 10.1016/j.jlr.2021.100125

Year: 2021

Cytokine-Mediated Regulation of ARG1 in Macrophages and Its Impact on the Control of Salmonella enterica Serovar Typhimurium Infection

DOI: 10.3390/cells10071823

Year: 2021

Regulation of Th1 T Cell Differentiation by Iron via Upregulation of T Cell Immunoglobulin and Mucin Containing Protein-3 (TIM-3)

DOI: 10.3389/fimmu.2021.637809

Year: 2021

Dietary Iron Overload and Hfe−/− Related Hemochromatosis Alter Hepatic Mitochondrial Function

DOI: 10.3390/antiox10111818

Year: 2021

The haemochromatosis gene Hfe and Kupffer cells control LDL cholesterol homeostasis and impact on atherosclerosis development.

DOI: 10.1093/eurheartj/ehaa140

Year: 2020

What are the Advantages of HTS in Infectious Disease Research?

Are There Specific Hygiene Recommendations for Food Safety?

Can Climate Change Affect Food and Water Security?

How Can Zika Virus Be Prevented?

What are Commonly Used ACTs?

How Do Pathogens Enter the Host?

How Do Apicomplexan Parasites Infect Hosts?

How do Bacteria Cause Diseases?

How Do Epidemiologists Track Infectious Diseases?

What are the Symptoms of ARDS?

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